Download PDF. 1 / 3 Pages. Previous article. Go back to website. Next article. HARRIS D., Paul BRINCK M., Patricio. Un análisis retrospectivo de cavernomatosis de la vena porta en Chile: II. Hemorragia, tratamiento y pronóstico. Rev. chil. HARRIS D., Paul BRINCK M., Patricio. Un análisis retrospectivo de cavernomatosis de la vena porta en Chile.: I. Perfil clínico y de laboratorio. Rev. chil. pediatr.

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Improved results of liver transplantation in patients with portal vein thrombosis. Complications of venous reconstruction pprta human orthotopic liver transplantation. Treatment with alteplase and heparin or with heparin alone in 10 patients] Gastroenterol Clin Biol. Transjugular intrahepatic portosystemic shunt.

Portal vein thrombosis: Insight into physiopathology, diagnosis, and treatment

Thrombolytic treatment of portal thrombosis. Occasionally, it is not possible to recognize any overt cause of PVT; generally, the clinical course is favorable for these patients, with a low incidence of complications. Cavernous transformation of the portal vein: Hepatobiliary Pancreat Dis Int. Liver transplantation in adults. In addition, MR angiography has a high accuracy in the follow-up of the portal venous system before and after liver transplantation[ 82 – 85 ].

The aetiology, presentation and natural history of extra-hepatic portal venous obstruction. N Engl J Med.

Anticardiolipin antibodies in patients with liver disease. Septic thrombophlebitis of the portal vein pylephlebitis: Incidence, risk factors, management, and outcome of portal vein abnormalities at orthotopic cavrnoma transplantation. However, in case of long standing thrombosis, the management of complications related to portal hypertension and portal cholangiopathy has to be concurrently considered[ ].

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The role of anticoagulation in pylephlebitis. Bacteroides species and thrombotic events, such as PVT[ 40 – 43 ]. Several causes can be involved in the pathogenesis of PVT and, frequently, more than one coexist. Nat Clin Pract Gastroenterol Hepatol.


Magnetic resonance portal venography: Unfortunately, in practice, this policy is not applicable without difficulty. PVT can be classified into four categories, depending on the extension: If no local risk factor is found, the presence of a thrombophilic disorder must be investigated.

Early development of cavernomatous vasculatures in portal venous thrombosis: In contrast, chronic PVT can be completely asymptomatic, or characterized portx splenomegaly, pancytopenia, varices, and, rarely, ascites[ 3 ].

Other approaches, such as transjugular intrahepatic portosystemic shunt placement, should be reserved for patients developing acute PVT before or after liver transplantation, or in alternative to thrombolysis when anticoagulation fails[]. If thrombosis is recent and there is no underlying thrombophilic condition, anticoagulation should be administered for mo, as a complete portal vein recanalization can occasionally be delayed[ 79,cxvernoma ].

Local or systemic pro-thrombotic factors, alone or together, can play an important role in PVT pathogenesis, which is complex and different in each clinical context and in each patient.

Portal vein thrombosis: Insight into physiopathology, diagnosis, and treatment

The goal of treatment is similar in acute and chronic PVT, and consists in correction of causal factors, prevention of thrombosis extension, and achievement of portal vein patency. Several studies reported a more complex surgical procedure, with a greater requirement of blood transfusions, an increased risk of complications such as primary non function or dysfunction, thrombosis of the hepatic artery, relaparotomy, postoperative pancreatitis, sepsis, caveenoma renal failurea poorer survival, and a higher mortality[ – ].

Incidentally, US is less reliable in determining the extension of the thrombus ds the mesenteric circulation[ 79 ]. Complete thrombosis of portal vein and proximal and distal superior mesenteric vein. HPB Oxford ; 9: Other less common PVT local causes are adenopathy, systemic inflammatory response syndrome, and surgical traumas to the portal venous system, such as portosystemic shunting, splenectomy, liver transplantation, ablative therapy for HCC, and venq needle aspiration of abdominal masses[ 1 ].


Inherited prothrombotic defects in Budd-Chiari syndrome and portal vein thrombosis: It seems to be prota in resolving portal biliopathy, ascites, and portal hypertension, but it is not feasible if portal vein is not catheterizable or a cavernomatous vein cannot be dilated[ – ]. National Center for Biotechnology InformationU.

Malformación cavernomatosa de la vena porta

Moreover, MRI-true fast imaging with steady state precession true FISPmight overcome the difficulty of contrast injection in cases of poor venous access and the degradation of the images by respiratory motion[ 86 ].

A population-based autopsy study. Portal vein grafts in hepatic transplantation.

Deficiency of natural anticoagulant proteins C, S, and antithrombin in portal vein thrombosis: In this review, we analyze the physiopathological mechanisms of PVT development, together with the hemodynamic and functional alterations related to this condition. Surgical thrombectomy is usually not recommended, as high morbidity and mortality have been reported; percutaneous transhepatic mechanical thrombectomy might also be effective in recent thrombosis, but vascular traumas are frequent and may stimulate rethrombosis[ ].